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  • 1.
    Vesikari, Timo
    et al.
    Univ Tampere, Sch Med, Vaccine Res Ctr, Biokatu 10, Tampere 33520, Finland..
    Richardus, Jan Hendrik
    Municipal Publ Hlth Serv GGD, Dept Infect Dis Control, Rotterdam, Netherlands..
    Berglund, Johan
    Blekinge Institute of Technology, Faculty of Engineering, Department of Health. Blekinge Inst Technol, Sch Hlth Sci, Karlskrona, Sweden..
    Korhonen, Tiina
    Univ Tampere, Sch Med, Vaccine Res Ctr, Biokatu 10, Tampere 33520, Finland..
    Flodmark, Carl-Erik
    Skane Univ Hosp, Childhood Obes Unit, Malmo, Sweden..
    Lindstrand, Ann
    Karolinska Inst, Dept Publ Hlth Sci, Stockholm, Sweden..
    Silfverdal, Sven Arne
    Umea Univ, Dept Clin Sci, Umea, Sweden..
    Bambure, Vinod
    Quanticate, Bangalore, Karnataka, India..
    Caplanusi, Adrian
    GSK Vaccines, Wavre, Belgium..
    Dieussaert, Ilse
    GSK Vaccines, Rixensart, Belgium..
    Roy-Ghanta, Sumita
    GSK Vaccines, King Of Prussia, PA USA..
    Vaughn, David W.
    GSK Vaccines, Rixensart, Belgium..
    Immunogenicity and Safety of a Trivalent Inactivated Influenza Vaccine in Children 6 Months to 17 Years of Age, Previously Vaccinated with an AS03-Adjuvanted A(H1N1)Pdm09 Vaccine Two Open-label, Randomized Trials2015In: The Pediatric Infectious Disease Journal, ISSN 0891-3668, E-ISSN 1532-0987, Vol. 34, no 7, p. 774-782Article in journal (Refereed)
    Abstract [en]

    Background: During the influenza pandemic 2009-2010, an AS03-adjuvanted A(H1N1) pdm09 vaccine was used extensively in children 6 months of age and older, and during the 2010-2011 influenza season, the A(H1N1) pdm09 strain was included in the seasonal trivalent inactivated influenza vaccine (TIV) without adjuvant. We evaluated the immunogenicity and safety of TIV in children previously vaccinated with the AS03-adjuvanted A(H1N1) pdm09 vaccine. Methods: Healthy children were randomized (1:1) to receive TIV or a control vaccine. Children were aged 6 months to 9 years (n = 154) and adolescents 10-17 years (n = 77) when they received AS03-adjuvanted A(H1N1) pdm09 vaccine at least 6 months before study enrolment. Hemagglutination inhibition (HI) and neutralizing antibody responses against the A(H1N1) pdm09 strain were evaluated before (day 0) and at day 28 and month 6 after study vaccination. Reactogenicity was assessed during the 7 day postvaccination period, and safety was assessed for 6 months. Results: At day 0, >93.9% of all children had HI titers >= 1:40 for the A(H1N1) pdm09 strain, which increased to 100% at both day 28 and month 6 in the TIV group. Between days 0 and 28, HI antibody geometric mean titers against A(H1N1) pdm09 increased by 9-fold and 4-fold in children 6 months to 9 years of age and 10-17 years of age, respectively. Conclusion: AS03-adjuvanted A(H1N1) pdm09 vaccine-induced robust immune responses in children that persisted into the next season, yet were still boosted by TIV containing A(H1N1) pdm09. The reactogenicity and safety profile of TIV did not appear compromised by prior receipt of AS03adjuvanted A(H1N1) pdm09 vaccine.

  • 2.
    Wilhelmsson, Peter
    et al.
    Linkoping Univ, Dept Clin & Expt Med, Med Microbiol, S-58185 Linkoping, Sweden..
    Fryland, Linda
    Linkoping Univ, Dept Clin & Expt Med, Infect Dis, S-58185 Linkoping, Sweden.;Linkoping Univ, Dept Clin & Expt Med, Clin Immunol, S-58185 Linkoping, Sweden..
    Lindblom, Pontus
    Linkoping Univ, Dept Clin & Expt Med, Med Microbiol, S-58185 Linkoping, Sweden..
    Sjowall, Johanna
    Linkoping Univ, Dept Infect Dis, S-58185 Linkoping, Sweden.;Linkoping Univ, Dept Clin & Expt Med, S-58185 Linkoping, Sweden..
    Ahlm, Gas
    Umea Univ, Dept Clin Microbiol, Div Infect Dis, S-90185 Umea, Sweden..
    Berglund, Johan
    Blekinge Institute of Technology, Faculty of Health Sciences, Department of Health.
    Haglund, Mats
    Kalmar Cty Hosp, Dept Infect Dis, S-38195 Kalmar, Sweden..
    Henningsson, Anna J.
    Ryhov Cty Hosp, Dept Clin Microbiol, Div Med Serv, S-55185 Jonkoping, Sweden..
    Nolskog, Peter
    Reg Vastra Gotaland Skaraborg Hosp, Dept Communicable Dis Control & Prevent, S-54185 Skovde, Sweden..
    Nordberg, Marika
    Aland Grp Borrelia Res, Mariehamn, Aland, Finland..
    Nyberg, Clara
    Aland Grp Borrelia Res, Mariehamn, Aland, Finland..
    Ornstein, Katharina
    Hassleholm Hosp, Dept Internal Med, S-28138 Hassleholm, Sweden.;Lund Univ, Dept Clin Sci, S-22100 Lund, Sweden..
    Nyman, Dag
    Aland Grp Borrelia Res, Mariehamn, Aland, Finland..
    Ekerfelt, Christina
    Linkoping Univ, Dept Clin & Expt Med, Clin Immunol, S-58185 Linkoping, Sweden.;Linkoping Univ, Dept Clin & Expt Med, S-58185 Linkoping, Sweden..
    Forsberg, Pia
    Linkoping Univ, Dept Clin & Expt Med, Infect Dis, S-58185 Linkoping, Sweden.;Linkoping Univ, Dept Infect Dis, S-58185 Linkoping, Sweden..
    Lindgren, Per-Eric
    Linkoping Univ, Dept Clin & Expt Med, Med Microbiol, S-58185 Linkoping, Sweden..
    A prospective study on the incidence of Borrelia burgdorferi sensu lato infection after a tick bite in Sweden and On the Aland Islands, Finland (2008-2009)2016In: Ticks and Tick-borne Diseases, ISSN 1877-959X, E-ISSN 1877-9603, Vol. 7, no 1, p. 71-79Article in journal (Refereed)
    Abstract [en]

    Lyme borreliosis (LB) is a common and increasing tick-borne disease in Europe. The risk of acquiring a Borrelia infection after a tick bite is not fully known. Therefore, we investigated the incidence of Borrelia infection after a bite by a Borrelia-infected tick and if the Borrelia load and/or the duration of tick-feeding influenced the risk of infection. During 2008-2009, ticks and blood samples were collected from 1546 tick-bitten persons from Sweden and the Aland Islands, Finland. Follow-up blood samples were taken 3 months after the tick bite. The duration of tick feeding was microscopically estimated and Borrelia was detected and quantified in ticks by real-time PCR. Anti-Borrelia antibodies were detected in sera using ELISA tests and immunoblot. Five percent (78/1546) of the study participants developed Borrelia infection (LB diagnosis and/or seroconversion) after a tick bite (45% bitten by Borrelia-infected ticks and 55% bitten by uninfected ticks). Of these, 33 developed LB (whereof 9 also seroconverted) while 45 participants seroconverted only. Experience of non-specific symptoms was more frequently reported by Borrelia-infected participants compared to uninfected participants. All who seroconverted removed "their" ticks significantly later than those who did not. The Borrelia load in the ticks did not explain the risk of seroconversion. Regional and sex differences in the Borrelia seroprevalence were found. The risk of developing a Borrelia infection after a bite by a Borrelia-infected tick is small but increases with the duration of tick feeding. (C) 2015 Elsevier GmbH. All rights reserved.

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